For decades, scientists have focused on beta-amyloid plaques as the primary cause of Alzheimer’s disease. These protein deposits were thought to disrupt cellular communication, leading to cognitive decline and brain cell death. However, the new study suggests that beta-amyloid may serve as a scaffolding for other proteins, which could be the true culprits behind brain damage in Alzheimer’s patients. Researchers at the Center for Neurodegenerative Disease have identified over 20 proteins capable of co-accumulating with beta-amyloid. This discovery challenges the linear amyloid cascade hypothesi…
